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Table 3 Haemostatic treatments and amounts of transfusion among patients with and without hyperfibrinolysis

From: Hyperfibrinolysis in severe isolated traumatic brain injury may occur without tissue hypoperfusion: a retrospective observational multicentre study

 

TBI

P value

Non-TBI

P value

Hyperfibrinolysis (-) n = 77

Hyperfibrinolysis (+) n = 34

Hyperfibrinolysis (-) n = 80

Hyperfibrinolysis (+) n = 46

Haemostatic intervention, n (%)

1 (1)

0 (0)

0.504

28 (35)

25 (54)

0.034

Tranexamic acid administration, n (%)

28 (36)

14 (41)

0.630

10 (13)

16 (35)

0.003

Transfusion in the first 6 h after admission

 RBC, units

0 (0–0)

0 (0–4)

<0.001

0 (0–0)

4 (0–12)

<0.001

FFP, units

0 (0–0)

0 (0–6)

<0.001

0 (0–0)

0 (0–10)

0.001

 PC, units

0 (0–0)

0 (0–0)

0.168

0 (0–0)

0 (0–0)

0.06

  ≥ 10 units RCC, n (%)

0 (0)

5 (15)

0.001

7 (9)

15 (33)

0.001

Transfusion in the first 24 h after admission

 RBC, units

0 (0–0)

0 (0–8)

<0.001

0 (0–0)

6 (0–16)

<0.001

 FFP, units

0 (0–0)

0 (0–6)

<0.001

0 (0–0)

4 (5–12)

<0.001

 PC, units

0 (0–0)

0 (0–0)

0.137

0 (0–0)

0 (0–0)

0.05

  ≥ 10 units RCC, n (%)

0 (0)

8 (20)

<0.001

9 (11)

16 (35)

0

  1. RBC red blood cell concentrate, FFP fresh frozen plasma, PC platelet concentrate, TBI traumatic brain injury